Aspirin inhibits the cyclooxygenase and thus the prostaglandin synthesis. The drug has analgesic, anti-inflammatory and antipyretic effects. Through its effect on the thrombocyte-cyclooxygenase it inhibits the formation of a highly effective platelet (thrombocyte) aggregator and a vasoconstrictor (thromboxane A2). Since the platelets do not synthesize proteins, the effect remains demonstrable as long as the affected thrombocytes live (7-10 days).

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